By Victor R. Preedy
Aging: Oxidative rigidity and nutritional Antioxidants bridges the trans-disciplinary divide and covers in one quantity the technology of oxidative tension in getting older and the doubtless healing use of common antioxidants within the nutrition or nutrients matrix. The approaches in the technology of oxidative pressure are defined in live performance with different tactics, equivalent to apoptosis, phone signaling, and receptor mediated responses. This technique acknowledges that illnesses are usually multifactorial, and oxidative pressure is a unmarried element of this.
Gerontologists, geriatricians, nutritionists, and dieticians are separated by way of divergent talents disciplines that have to be bridged so that it will boost preventative in addition to remedy innovations. whereas gerontologists and geriatricians may possibly learn the underlying strategies of getting older, they're much less more likely to be acquainted with the technology of meals and dietetics. however, nutritionists and dietitians are much less conversant with the distinct scientific heritage and technological know-how of gerontology. This ebook addresses this hole and brings each one of those disciplines to endure at the methods inherent within the oxidative tension of getting older.
- Nutritionists can follow details regarding mitochondrial oxidative pressure in a single ailment to diet-related techniques in one other unrelated illness
- Dietitians can prescribe new meals or diets containing anti-oxidants for stipulations immune to traditional pharmacological remedies
- Dietitians, after studying concerning the simple biology of oxidative rigidity, could be in a position to recommend new remedies to their multidisciplinary teams
- Nutritionists and dietitians will achieve an realizing of mobile signaling and be capable to recommend new preventative or healing recommendations with anti-oxidant wealthy foods
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Additional info for Aging. Oxidative Stress and Dietary Antioxidants
The longevity network principally refers to positive and negative feedback mechanisms between four genes: SIRT1, IGF1, AMPK and mTOR. The signaling between these genes regulates biologic aging and numerous age-related diseases, including cardiovascular diseases. At the end of each line, an arrowhead indicates positive regulation/stimulation, and a round end indicates negative regulation or inhibition. SIRT1: Sirtuin1; TSC1/2: tuberous sclerosis 1/2; UCP2: mitochondrial uncoupling protein 2; IRS2: insulin receptor substrate 2; FOXO: O class forkhead box transcription factor; IGF1: insulin-like growth factor 1; AKT; protein kinase B; TOR/mTOR: target of rapamycin/mammalian target of rapamycin; ERK1/2: extracellular signal-regulated kinases 1/2; AMPK: adenosine monophosphate-activated protein kinase; Nampt: nicotinamide phosphoribosyltransferase; NADH: nicotinamide adenine dinucleotide; LKB: liver kinase B.
N,N-Dimethyl-p-phenylenediamine dihydrochloride-based method for the measurement of plasma oxidative capacity during human aging. Anal Biochem 2013;436:165–7. 032. Antelmi I, de Paula RS, Shinzato AR, et al. Influence of age, gender, body mass index, and functional capacity on heart rate variability in a cohort of subjects without heart disease. Am J Cardiol 2004;93:381–5. Cosentino F, Francia P, Camici GG, et al. Final common molecular pathways of aging and cardiovascular disease: role of the p66Shc protein.
Cardiovasc Res 2000;47:446–56. Vazquez-Medina JP, Popovich I, Thorwald MA, et al. Angiotensin receptor-mediated oxidative stress is associated with impaired cardiac redox signaling and mitochondrial function in insulin- resistant rats. Am J Physiol Heart Circ Physiol 2013;305:H599–607. 2013. Qin F, Lennon-Edwards S, Lancel S, et al. Cardiac-specific overexpression of catalase identifies hydrogen peroxide-dependent and -independent phases of myocardial remodeling and prevents the progression to overt heart failure in G(alpha)q-overexpressing transgenic mice.