Advances in Comparative and Environmental Physiology: by F. Lang, M. Ritter, H. Völkl, D. Häussinger (auth.), Prof.

By F. Lang, M. Ritter, H. Völkl, D. Häussinger (auth.), Prof. Dr. Florian Lang, Prof. Dr. Dieter Häussinger (eds.)

One of the necessities for survival is the facility of cells to take care of their quantity. therefore, in the course of the process evolution cells have "learned" numerous suggestions to accomplish quantity homeostasis. This quantity regulatory equipment consists of legislation of either, mobile metabolism and mobile shipping and is exploited through hormones and transmitters to manage cellularfunction. This booklet to illustrates the complicated interaction of telephone quantity regulatory mechanisms and mobile functionality in various tissues. although, our wisdom continues to be faraway from being conclusive, and the current selection of stories is assumed to foster extra experimental efforts to resolve the function of mobile quantity within the built-in functionality of cells.

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Hiiussinger 1 , W. Gerokl, and F. Lang2 Contents 1 Introduction. . . . . . . . . . . . . . . . . . . . . . . . . . . . . 2 Cell Volume Control in Liver ............................................ 1 Anisoosmotic Cell Volume Modulation ................................... 2 Cumulative Substrate Transport ......................................... 3 Cell Volume Control by Hormones ....................................... 3 Modulation of Hepatic Metabolism by Cell Volume ........................

G. result in marked intracellular accumulation of (almost impermeable) glutamate as soon as urea synthesis is blocked. 3 Cell Volume Control by Hormones Among other actions, hormones are known to modify the activities of ion transporters and channels in the plasma membrane, to affect the cell membrane potential and to modulate Na + -driven substrate transport (Fehlmann and Freychat 1981; Fehlmann et al. 1981; Lynch et al. 1986; Jakubowski and Jakob 1990); for review, see Moule and McGivan 1990).

1991c; Fig. 3). Accordingly, one is tempted to speculate that physiological fluctuations of the portal insulin and glucagon concentrations may modify liver cell volume also in vivo. In perfused rat liver, vasopressin exhibits only minor effects on cell volume, but shrinks the cells when added on top of glucagon or cAMP (vom Dahl et al. 1991c). Adenosine leads to cell shrinkage, whereas phenylephrine slightly increases liver cell volume. The effects of a variety of hormones on liver cell volume are summarized in Table 1.

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